Pharmacologic down-regulation of EZH2 suppresses bladder cancer in vitro and in vivo

نویسندگان

  • Shou-Hung Tang
  • Hsu-Shan Huang
  • Hong-Ui Wu
  • Yi-Ta Tsai
  • Mei-Jen Chuang
  • Cheng-Ping Yu
  • Shih-Ming Huang
  • Guang-Huan Sun
  • Sun-Yran Chang
  • Pei-Wen Hsiao
  • Dah-Shyong Yu
  • Tai-Lung Cha
چکیده

The polycomb group gene, EZH2, is highly expressed in advanced bladder cancer. Here we demonstrated that down-regulation of EZH2 in tumor tissues after neo-adjuvant chemotherapy correlated with good therapeutic response in advanced bladder cancer. We next developed a small molecule, NSC745885, derived from natural anthraquinone emodin, which down-regulated EZH2 via proteasome-mediated degradation. NSC745885 showed potent selective toxicity against multiple cancer cell lines but not normal cells. NSC745885 treatment overcame multiple-drug resistance and inhibited growth of resistant cancer cells. Over-expression of EZH2 in cancer cells attenuated effects of NSC745885, suggesting that down-regulation of EZH2 was responsible for growth inhibition of NSC745885. NSC745885 also suppressed tumor growth and down-regulated EZH2 in vivo. These results indicate that NSC7455889 suppresses bladder cancer by targeting EZH2.

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عنوان ژورنال:

دوره 5  شماره 

صفحات  -

تاریخ انتشار 2014